Sleep Apnea and phentermine
sleep Apnea and
phentermine, Getting full nights sleep, and having little to no
sweating from night sweats, without the aid of drugs will help in allowing
other medications to accomplish their tasks. Your body will respond to
treatments faster when you are well rested. sleep Apnea and phentermine is a
problem and if you can avoid night sweats naturally you will be better of in
the long run. being that sweating ant night is a sleep apnea symptom, you
should read this entire web site from the start to get a full
understanding.
Until you have a sleep apnea cure, you should try to be as comfortable as
you can.
Sleep Apnea and Phentermine
Sleep Apnea
- Obesity, particularly upper body obesity, is the most significant risk factor for obstructive sleep apnea.
- There is a 12 to 30-fold higher incidence of obstructive sleep apnea among morbidly obese patients compared to the general population.
- Among patients with obstructive sleep apnea, at least 60% to 70% are obese
- Phentermine is a drug that was part of the Fen-phen anti-obesity drug, the other drug being fenfluramine. Fenfluramine was withdrawn from the U.S. market in 1997 after reports of heart valve disease and pulmonary hypertension.
Phentermine is still available by itself in most countries, including the U.S. Internationally, phentermine is a Schedule IV drug under the Convention on Psychotropic Substances[1].
Phentermine, like many other prescription drugs, work with neurotransmitters in the brain. It stimulates neuron bundles to release a particular group of neurotransmitters known as catecholamines; these include dopamine, epinephrine (formally known as adrenalin), and norepinephrine (noradrenalin). They signal a fight-or-flight response in your body which, in turn, puts a halt to the hunger signal. As a result, you lose your appetite because your brain doesn’t receive the hunger message. This is perhaps due to phentermine’s affects on leptin levels in the brain. It is theorized that phentermine can raise levels of leptin which signal satiety. It is also theorized that increased levels of the catecholamines are partially responsible for halting another chemical messenger known as neuropeptide Y. This chemical initiates eating, decreases energy expenditure, and increases fat storage.
